Zeitschriftenaufsatz | 2006 Open Access

STAT1 acts as a tumor promoter for leukemia development.

Autor:in

Kovacic, Boris; Stoiber, Dagmar; Moriggl, Richard; Weisz, Eva; Ott, René G; Kreibich, Rita; Levy, David E; Beug, Hartmut; Freissmuth, Michael; Sexl, Veronika;

Publikationen als Autor:in / Herausgeber:in der Vetmeduni

Sexl, Veronika

Journal

Cancer Cell

Schlagwörter

Animals; B-Lymphocytes/metabolism; B-Lymphocytes/pathology; Cell Line, Tumor; Cell Proliferation; Cell Survival/genetics; Cell Transformation, Neoplastic/genetics; DNA-Binding Proteins/genetics; DNA-Binding Proteins/metabolism; Disease Progression; Genotype; Histocompatibility Antigens Class I/immunology; Histocompatibility Antigens Class I/metabolism; Interferon-gamma/genetics; Interferon-gamma/metabolism; Killer Cells, Natural/immunology; Killer Cells, Natural/metabolism; Leukemia, Experimental/genetics; Leukemia, Experimental/metabolism; Leukemia, Experimental/pathology*; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; Mice, Knockout; Oncogene Proteins v-abl/genetics; Oncogene Proteins v-abl/metabolism; Oncogene Proteins, Fusion/genetics; Oncogene Proteins, Fusion/metabolism; Phenotype; STAT1 Transcription Factor/deficiency; STAT1 Transcription Factor/genetics; STAT1 Transcription Factor/physiology*; Stem Cells/metabolism; Stem Cells/pathology; Survival Analysis;

Dokumententyp

Originalarbeit

ISSN/eISSN

1535-6108 -

DOI

10.1016/j.ccr.2006.05.025

WoS ID

WOS:000239202800010

PubMed ID

16843267