Zeitschriftenaufsatz
|
2011
High-affinity IgE receptors on dendritic cells exacerbate Th2-dependent inflammation.
Autor:in
Sallmann, Eva; Reininger, Bärbel; Brandt, Sabine; Duschek, Nikolaus; Hoflehner, Elisabeth; Garner-Spitzer, Erika; Platzer, Barbara; Dehlink, Eleonora; Hammer, Martina; Holcmann, Martin; Oettgen, Hans C; Wiedermann, Ursula; Sibilia, Maria; Fiebiger, Edda; Rot, Antal; Maurer, Dieter;
Publikationen als Autor:in / Herausgeber:in der Vetmeduni
Journal
Schlagwörter
Allergens/toxicity; Animals; Antigens, Plant/toxicity; Cells, Cultured; Coculture Techniques; Dendritic Cells/immunology*; Dendritic Cells/metabolism; Dendritic Cells/pathology*; Female; Humans; Inflammation Mediators/metabolism*; Inflammation Mediators/physiology; Inflammation Mediators/toxicity; Male; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; Mice, Knockout; Mice, Transgenic; Ovalbumin/toxicity; Protein Binding/genetics; Protein Binding/immunology; Receptors, IgE/deficiency; Receptors, IgE/metabolism*; Receptors, IgE/physiology; Th2 Cells/immunology*; Th2 Cells/metabolism; Th2 Cells/pathology*; Time Factors;
Dokumententyp
Originalarbeit
ISSN/eISSN
0022-1767 -
10.4049/jimmunol.1003392
WoS ID
PubMed ID