Zeitschriftenaufsatz | 2012 Open Access

Type I interferons promote fatal immunopathology by regulating inflammatory monocytes and neutrophils during Candida infections.

Autor:in

Majer, Olivia; Bourgeois, Christelle; Zwolanek, Florian; Lassnig, Caroline; Kerjaschki, Dontscho; Mack, Matthias; Müller, Mathias; Kuchler, Karl;

Publikationen als Autor:in / Herausgeber:in der Vetmeduni

Müller, Mathias; Lassnig, Caroline

Journal

PLoS Pathogens

Schlagwörter

Animals; Antigens, CD/biosynthesis; Antigens, Ly/biosynthesis; Candida albicans/immunology*; Candidemia/mortality; Candidiasis/immunology*; Candidiasis/pathology; Chemokine CCL2/biosynthesis; Chemokine CXCL1/biosynthesis; Dendritic Cells/immunology; Inflammation/drug therapy; Inflammation/immunology; Interferon Type I/metabolism*; Kidney/immunology; Kidney/microbiology; Male; Membrane Glycoproteins/biosynthesis; Mice; Mice, Inbred C57BL; Mice, Knockout; Monocytes/drug effects; Monocytes/immunology*; Neutrophils/drug effects; Neutrophils/immunology*; Nitric Oxide Synthase Type II/biosynthesis; PPAR gamma/agonists; Receptors, Interferon/deficiency; Receptors, Interferon/genetics; Signal Transduction/genetics; Thiazolidinediones/pharmacology; Thiazolidinediones/therapeutic use;

Dokumententyp

Originalarbeit

ISSN/eISSN

1553-7366 -

DOI

10.1371/journal.ppat.1002811

WoS ID

WOS:000306837700032

PubMed ID

22911155

Titel des Sammelwerks

PLOS PATHOGENS;