Cooperation of Th1 and Th17 cells determines transition from autoimmune myocarditis to dilated cardiomyopathy.

Autor:in

Nindl, Veronika; Maier, Reinhard; Ratering, David; De Giuli, Rita; Züst, Roland; Thiel, Volker; Scandella, Elke; Di Padova, Franco; Kopf, Manfred; Rudin, Markus; Rülicke, Thomas; Ludewig, Burkhard;

Schlagwörter

Animals; Autoantigens/immunology; Autoimmune Diseases/immunology*; Autoimmune Diseases/pathology; Cardiomyopathy, Dilated/immunology*; Cardiomyopathy, Dilated/pathology*; Disease Models, Animal; Heart Ventricles/immunology; Heart Ventricles/pathology; Inflammation/immunology; Inflammation/pathology; Interferon-gamma/immunology; Interleukin-17/immunology; Male; Mice; Mice, Inbred BALB C; Mice, Transgenic; Myocarditis/immunology*; Myocarditis/pathology*; Receptors, Antigen, T-Cell/immunology; Th1 Cells/immunology*; Th17 Cells/immunology*; Ventricular Remodeling/immunology;

ISSN/eISSN

0014-2980 -

WoS ID

WOS:000308404800014