The CD4(+) AT2R(+) T cell subpopulation improves post-infarction remodelling and restores cardiac function.

Autor:in

Skorska, Anna; von Haehling, Stephan; Ludwig, Marion; Lux, Cornelia A; Gaebel, Ralf; Kleiner, Gabriela; Klopsch, Christian; Dong, Jun; Curato, Caterina; Altarche-Xifró, Wassim; Slavic, Svetlana; Unger, Thomas; Steinhoff, Gustav; Li, Jun; David, Robert;

Schlagwörter

Animals; CD4-Positive T-Lymphocytes/immunology*; Cardiotonic Agents/metabolism; Heart Failure/blood; Heart Failure/complications; Heart Failure/immunology; Heart Failure/physiopathology; Heart Function Tests*; Humans; Immunomodulation; Interleukin-10/blood; Myocardial Infarction/blood; Myocardial Infarction/complications; Myocardial Infarction/immunology*; Myocardial Infarction/physiopathology*; Myocardial Ischemia/blood; Myocardial Ischemia/complications; Myocardial Ischemia/immunology; Myocardial Ischemia/physiopathology; Rats, Wistar; Receptor, Angiotensin, Type 2/metabolism*; Tumor Necrosis Factor-alpha/blood; Ventricular Remodeling*;

CC Lizenz

CCBY

ISSN/eISSN

1582-1838 -

WoS ID

WOS:000358925100022