TRIF signaling drives homeostatic intestinal epithelial antimicrobial peptide expression.

Autor:in

Stockinger, Silvia; Duerr, Claudia U; Fulde, Marcus; Dolowschiak, Tamas; Pott, Johanna; Yang, Ines; Eibach, Daniel; Bäckhed, Fredrik; Akira, Shizuo; Suerbaum, Sebastian; Brugman, Martijn; Hornef, Mathias W;

Schlagwörter

Adaptor Proteins, Vesicular Transport/genetics; Adaptor Proteins, Vesicular Transport/immunology*; Animals; Antimicrobial Cationic Peptides/biosynthesis; Antimicrobial Cationic Peptides/immunology*; Cyclins/metabolism; Immunity, Innate/immunology*; Intestinal Mucosa/immunology*; Intestinal Mucosa/microbiology; Listeria monocytogenes/immunology*; Listeriosis/immunology; Mice; Mice, Inbred C57BL; Mice, Knockout; Myeloid Differentiation Factor 88/genetics; Paneth Cells/metabolism; Proteins/metabolism*; Receptors, Interferon/genetics; Salmonella Infections/immunology; Salmonella typhimurium/immunology*; Signal Transduction/immunology; Toll-Like Receptor 3/genetics; Toll-Like Receptor 4/genetics;

ISSN/eISSN

0022-1767 -

WoS ID

WOS:000344078200040