Zeitschriftenaufsatz
|
2014
Co-operating STAT5 and AKT signaling pathways in chronic myeloid leukemia and mastocytosis: possible new targets of therapy.
Autor:in
Bibi, Siham; Arslanhan, Melis Dilara; Langenfeld, Florent; Jeanningros, Sylvie; Cerny-Reiterer, Sabine; Hadzijusufovic, Emir; Tchertanov, Luba; Moriggl, Richard; Valent, Peter; Arock, Michel;
Publikationen als Autor:in / Herausgeber:in der Vetmeduni
Schlagwörter
Animals; Fusion Proteins, bcr-abl/genetics; Fusion Proteins, bcr-abl/metabolism; Humans; Janus Kinases/metabolism; Leukemia, Myelogenous, Chronic, BCR-ABL Positive/drug therapy; Leukemia, Myelogenous, Chronic, BCR-ABL Positive/etiology; Leukemia, Myelogenous, Chronic, BCR-ABL Positive/metabolism*; Mast Cells/metabolism; Mast Cells/pathology; Mastocytosis/drug therapy; Mastocytosis/etiology; Mastocytosis/metabolism*; Molecular Targeted Therapy; Mutation; Phosphatidylinositol 3-Kinases/metabolism; Proto-Oncogene Proteins c-akt/antagonists & inhibitors; Proto-Oncogene Proteins c-akt/metabolism*; Proto-Oncogene Proteins c-kit/genetics; Proto-Oncogene Proteins c-kit/metabolism; STAT5 Transcription Factor/antagonists & inhibitors; STAT5 Transcription Factor/metabolism*; Signal Transduction*/drug effects;
Dokumententyp
Originalarbeit
CC Lizenz
CCBY
Open Access Type
Gold
ISSN/eISSN
0390-6078 -
10.3324/haematol.2013.098442
WoS ID
PubMed ID