Lack of Galanin Receptor 3 Alleviates Psoriasis by Altering Vascularization, Immune Cell Infiltration, and Cytokine Expression.

Autor:in

Locker, Felix; Vidali, Silvia; Holub, Barbara S; Stockinger, Julia; Brunner, Susanne M; Ebner, Sabine; Koller, Andreas; Trost, Andrea; Reitsamer, Herbert A; Schwarzenbacher, David; Lang, Roland; Kofler, Barbara

Schlagwörter

Adult; Animals; Cytokinesmetabolism; Disease Models, Animal; Female; Humans; Imiquimodimmunology; Keratinocytes; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; Middle Aged; Neovascularization, Pathologicgeneticspathology; Nestinmetabolism; Neutrophil Infiltration; Neutrophilsimmunology; Psoriasisdiagnosisgeneticsimmunologypathology; Receptor, Galanin, Type 2geneticsmetabolism; Receptor, Galanin, Type 3geneticsmetabolism; Severity of Illness Index; Skinblood supplydrug effectsimmunologypathology

ISSN/eISSN

0022-202X -

WoS ID

WOS:000418495900031