Zeitschriftenaufsatz | 2017

Cutting Edge: NKG2D Signaling Enhances NK Cell Responses but Alone Is Insufficient To Drive Expansion during Mouse Cytomegalovirus Infection.

Autor:in

Nabekura, Tsukasa; Gotthardt, Dagmar; Niizuma, Kouta; Trsan, Tihana; Jenus, Tina; Jonjic, Stipan; Lanier, Lewis L

Publikationen als Autor:in / Herausgeber:in der Vetmeduni

Journal

Schlagwörter

Adaptor Proteins, Signal Transducinggeneticsmetabolism; Animals; Cell Proliferation; Cells, Cultured; Herpesviridae Infectionsimmunology; Immunity, Innate; Killer Cells, Naturalimmunology; Lymphocyte Activation; Mice; Mice, Inbred C57BL; Mice, Knockout; Muromegalovirusimmunology; NK Cell Lectin-Like Receptor Subfamily Ageneticsmetabolism; NK Cell Lectin-Like Receptor Subfamily Kgeneticsmetabolism; Protein Binding; Receptors, Immunologicgeneticsmetabolism; Signal Transduction

Dokumententyp

Originalarbeit

ISSN/eISSN

0022-1767 -

DOI

10.4049/jimmunol.1700799

WoS ID

WOS:000408005200006

PubMed ID

28760883