Zeitschriftenaufsatz
|
2004
Cardiac myocyte-specific HIF-1alpha deletion alters vascularization, energy availability, calcium flux, and contractility in the normoxic heart.
Autor:in
Huang, Yan; Hickey, Reed P; Yeh, Jennifer L; Liu, Dinggang; Dadak, Agnes; Young, Lawrence H; Johnson, Randall S; Giordano, Frank J;
Publikationen als Autor:in / Herausgeber:in der Vetmeduni
Journal
Schlagwörter
Animals; Calcium Signaling/physiology*; Coronary Circulation/physiology*; DNA-Binding Proteins/deficiency; DNA-Binding Proteins/genetics; DNA-Binding Proteins/physiology*; Energy Metabolism; Gene Deletion; Gene Expression Regulation/physiology; Heart Function Tests; Hypoxia-Inducible Factor 1; Hypoxia-Inducible Factor 1, alpha Subunit; Mice; Mice, Inbred C57BL; Mice, Knockout; Myocardial Contraction/physiology*; Myocardium/metabolism*; Myocytes, Cardiac/physiology*; Neovascularization, Physiologic/physiology; Nuclear Proteins/deficiency; Nuclear Proteins/genetics; Nuclear Proteins/physiology*; Oxygen Consumption; RNA, Messenger/biosynthesis; Reverse Transcriptase Polymerase Chain Reaction; Transcription Factors/deficiency; Transcription Factors/genetics; Transcription Factors/physiology*; Transcription, Genetic;
Dokumententyp
Originalarbeit
ISSN/eISSN
0892-6638 -
10.1096/fj.04-1510fje
WoS ID
PubMed ID