Zeitschriftenaufsatz
|
2014
STAT1β is not dominant negative and is capable of contributing to gamma interferon-dependent innate immunity.
Autor:in
Semper, Christian; Leitner, Nicole R; Lassnig, Caroline; Parrini, Matthias; Mahlakõiv, Tanel; Rammerstorfer, Michael; Lorenz, Karin; Rigler, Doris; Müller, Simone; Kolbe, Thomas; Vogl, Claus; Rülicke, Thomas; Staeheli, Peter; Decker, Thomas; Müller, Mathias; Strobl, Birgit;
Publikationen als Autor:in / Herausgeber:in der Vetmeduni
Journal
Schlagwörter
Animals; Cell Nucleus/drug effects; Cell Nucleus/metabolism; Gene Expression Regulation/drug effects; Gene Knock-In Techniques; Genes, Dominant*; Herpesviridae Infections/immunology; Herpesviridae Infections/pathology; Immunity, Innate/drug effects*; Interferon-beta/pharmacology; Interferon-gamma/pharmacology*; Listeria/drug effects; Listeria/physiology; Listeriosis/immunology; Listeriosis/pathology; Mice; Muromegalovirus/drug effects; Muromegalovirus/physiology; Phosphorylation/drug effects; Phosphotyrosine/metabolism; Promoter Regions, Genetic/genetics; Protein Binding/drug effects; Protein Binding/genetics; Protein Isoforms/metabolism; Protein Transport/drug effects; STAT1 Transcription Factor/deficiency; STAT1 Transcription Factor/metabolism*; Transcription, Genetic/drug effects;
Dokumententyp
Originalarbeit
Open Access Type
Green
ISSN/eISSN
0270-7306 -
WoS ID
PubMed ID